在复合材料中由于制备工艺或后期损伤导致的弱结合界面，界面破坏为主要的复合材料破坏机制。
Interface breakage will be the main fracture mechanism if the interface bonding is weak because of the damage during the composites preparation.
酸所造成的破坏要弱于碱性物质，因为许多角膜蛋白质可结合酸并起到化学缓冲的作用。
Acids tend to cause less damage than alkalis as many corneal proteins bind acid and act as a chemical buffer.
新材料是超分子聚合物，由金属间弱相互作用力将小分子聚合而成，就如同一种分子尼龙扣的结合。
It's a supramolecular polymer, made of smaller molecules held together by weak interactions with metals, which act like a kind of molecular Velcro.
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