延胡索酸，反丁烯二酸，富马酸：fumaric acid 延迟遗传：delayed ingeritance 延迟性可逆性缺血性神经机能障碍：delayed reversible ischemic nervous dysfunction (DRIND) ..
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结论原发性高血压是由DNA损伤引起的人类表型缺陷，该病症符合常染色体显性遗传，具延迟外显性。
Conclusion: the essential hypertension is the human phenotype blemish caused by injured DNA. It is compatible with an autosomal dominant inheritance and its performance is delayed.
环境毒素或是遗传敏感性可能也是病因。药物疗法必须谨慎规划并结合耐药性与副作用的延迟作用。
Environmental toxins or genetic susceptibility may account for some cases. Drug therapy requires careful scheduling and combinations to delay development of tolerance and side effects.
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