尿毒症毒素通过影响线粒体功能而导致细胞损伤。
In CRF, uremic toxins injured cell by influencing mitochondria.
背景2011年5月在德国发生了一次由产志贺毒素大肠杆菌o 104: H4引起的溶血性尿毒症综合征的大暴发流行。
BACKGROUND a large outbreak of the hemolytic-uremic syndrome caused by Shiga-toxin-producing Escherichia coli O104: H4 occurred in Germany in May 2011.
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