这会导致胎盘的氧化应急和损伤，特别是对于合胞体滋养层。

This leads to oxidative stress and damage to the placenta, specifically to the syncytiotrophoblast.

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当合胞体滋养层细胞死亡，他们就会释放到母体的血液循环中，从而引起炎症级联反应，进而损害母体的器官。

When syncytiotrophoblast cells die, they are released into the maternal circulation, initiating a cascade of inflammation that can damage maternal organs.

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进一步的研究需要确定一氧化碳是否可以在动物模型中抑制合胞体滋养层的死亡，以及是否有和一氧化碳相似的途径存在保护作用。

Future studies will determine whether carbon monoxide can prevent syncytiotrophoblast death in animal models and whether other approaches similar to carbon monoxide may provide feasible protection.

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