三叶因子家族多肽的作用机理仍处于猜测阶段，现有与粘蛋白共同作用和与受体作用两种假说。
The mechanism of TFF is not clear and two hypotheses were proposed which were co working with mucin or receptor.
大多数雄激素非依赖性前列腺癌均高表达AR和核受体辅活化子、如转录中间因子2（TIF-2），ARA24、PIAS、类固醇受体辅活化子-1（SRC-1）等有力地支持了这个假说。
The "superactive AR"hypothesis was supported by the fact that the majority of AIPC expresses high level of AR and nuclear receptor(NR) coactivators, such as TIF-2, ARA24, PIAS, SRC-1.
到目前为上的假说是：脑内某些区域如海马中糖皮质激素受体的改变可能引起抑郁症状。
It has been hypothesized that changes in the glucocorticoid receptors in certain regions of the brain (eg, the hippocampus) might contribute to the depressive symptoms.
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