The paroxysmal depolarization shift formed by transient and quick inflow and slow inflow of Ca~(2+) was considered to be the basis of epileptic discharge. The ambulation of Ca~(2+) was completed by calcium channel.
目前认为短暂而快速的钙内流与缓慢的钙内流形成神经元的阵发性去极化漂移是痫性放电的基础。
参考来源 - 尼莫地平对青霉素致痫大鼠海马组织保护作用的研究·2,447,543篇论文数据，部分数据来源于NoteExpress
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