这暗示着卵母细胞成熟抑制因子靠酶切hax - 1蛋白来启动线粒体中的细胞凋亡。
This suggests that Omi can initiate apoptosis in the mitochondria by cleaving HAX-1 protein.
该试验清楚地表明了卵母细胞成熟抑制因子单独决定HAX - 1的酶切，在本实验使用的条件下对于细胞凋亡是必须的。
This experiment clearly shows that Omi is solely responsible for HAX-1 cleavage, which is essential for apoptosis under the conditions used in these experiments.
这和最近的研究是一致的，该研究表明卵母细胞成熟抑制因子能诱导用TNF处理的人类神经多肽的凋亡而不从线粒体中释放出来（7）。
This is in accord with a recent study that shows Omi can induce apoptosis in human neutrophils treated with TNF- without being released from the mitochondria (7).
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