It has been reported that a lot of hypothesis took part in carbon monoxide poisoning,such as ischemic-hypoxia、cell toxic injury、free radical formation、excitatory amino acids formation、apoptosis mechanism and so on.
有关CO中毒及其迟发性脑病发病机制的研究,到目前为止已提出了一些假说,如缺血缺氧、细胞毒性损伤、自由基的形成、兴奋性氨基酸的生成、炎症、微微血栓形成、凋亡机制等。
参考来源 - 动态研究CO中毒小鼠白细胞表面粘附分子CD11b/CD18、血小板CD61及脑组织病理改变·2,447,543篇论文数据，部分数据来源于NoteExpress
与其它肿瘤一样，骨肉瘤的发生与凋亡机制有关。
As the other tumors, the osteosarcomatous genesis is correlated with apoptosis mechanism.
其对其他DNA损伤诱导的细胞凋亡机制的作用还不清楚。
However, its role in other DNA-damage-induced apoptosis mechanisms is less well established.
最近的研究表明，凋亡机制参与了脑出血后神经细胞的死亡。
Recent studies have suggested that apoptosis involved in nerve cell death following ICH.
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