[摘要] 正 心肌和骨骼肌的缺血模型研究表明,多形核(PMN)白细胞是导致缺血再灌流(Reperfusion)后无再流(No-reflow)的主要原因。PMN 白细胞是否在脑缺血再灌流损伤中起重要作用,目前尚不甚清楚。
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These findings indicated that brain tissue injury persists,and may become even severe in the early stage of reperfusion.
本文实验结果表明再灌流早期脑组织损伤继续存在，甚至更为严重。
参考来源 - 脑缺血及再灌流对皮层体感诱发电位及神经细胞外钙离子活度的影响·2,447,543篇论文数据，部分数据来源于NoteExpress
结果表明较长时间缺血后再灌流加重组织损伤。
The results demonstrate that reperfusion after prolonged ischemia exaggerates tissue injury.
方法采用线栓法制备大脑中动脉缺血再灌流模型。
Methods The focal cerebral ischemia and reperfusion model was made by thread embolism of MCA.
方法采用线栓法制备大脑中动脉缺血再灌流模型。
Methods We made the local cerebral ischemic reperfusion model with thread embolism of MCA.
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