艾滋病毒是通过相关的免疫激活，慢性炎症，凝血功能障碍，和血脂紊乱机制，促进动脉粥样硬化形成。
HIV is known to promote atherosclerosis through mechanisms related to immune activation, chronic inflammation, coagulation disorders, and lipid disturbances.
游离二氧化硅致肺组织纤维化机制十分复杂，涉及多种细胞及生物活性物质参与的炎症反应、免疫反应、异物反应、细胞毒作用等。
The mechanism of fibrosis caused by free silica particles involves a lot of cells and reactions including inflammatory reaction, immunological reaction, foreign body reaction and cell toxic action.
探讨独活及其醇提物延缓脑老化的自由基和免疫炎症方面的作用机制，为延缓机体衰老提供实验依据。
Based upon free radicals and inflammation theory on senescense, this study further researches the mechanism in delaying the brain aging process by Angelica pubescens and its extractive.
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