生物细胞为了保持遗传物质DNA的稳定．具有种种结构．DNA修复酶就是其中之一。
因此，prx1的修复酶作为保持青春的关键酶浮出水面。
Repair of the peroxiredoxin Prx1 consequently emerges as a key process in aging.
绝大多数传统锯齿状腺瘤保留错配修复酶功能，表明微卫星稳定表型。
The overwhelming majority of traditional serrated adenomas retain mismatch repair enzyme function indicating a microsatellite-stable phenotype.
相反，我们如今已经可以推测这种prx1修复酶的增加是否能抵消至少延缓肿瘤的产生。
Conversely, we can now speculate whether increased repair of Prx1 during aging can counteract, or at least delay, the development of cancer.
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