目的：探讨幼龄鼠未成熟脑组织对惊厥性脑损伤中细胞凋亡与坏死病理过程具有主动保护性抑制机制的分子生物学机理。
Objective: to explore the molecular biological base of internal protective response in premature brain which reduces the process of apoptosis and necrosis of neurons.
近来的证据显示：自噬提供了一个保护性功能来限制肿瘤坏死和炎症，同时减轻了肿瘤细胞内的染色体损伤来回应代谢性应激。
Recent evidence suggests that autophagy provides a protective function to limit tumour necrosis and inflammation and to mitigate genome damage in tumour cells in response to metabolic stress.
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