...l Obesity with little Abdominal Subcutaneous Fat （二）内脏脂肪积聚是引起代谢综合征的关键因素 内脏肥胖(visceral obesity) 大网膜、肠系膜、腹膜后部 分泌几十种脂肪因子(adipokines), 参与信息传递、 调节胰岛素敏感性、影响糖脂代谢和能量平衡；调节 ...
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abdominal visceral obesity 腹型肥胖患者
Visceral fat obesity 内脏脂肪型肥胖
Masked visceral fat obesity 隐性内脏脂肪性肥胖
Pseudo visceral fat obesity 假性内脏脂肪性肥胖
3. The role of peripheral type of obesity on insulin resistance surpass that of visceral obesity, reach more than once.
3.周围型肥胖对胰岛素抵抗的作用大于中心性肥胖,是中心性肥胖的1倍以上。
参考来源 - 哈萨克族肥胖、胰岛素抵抗与糖调节异常的关系·2,447,543篇论文数据，部分数据来源于NoteExpress
This implies that adiponectin level is more correlated with visceral obesity than with whole body obesity.
提示与全身性肥胖相比，脂联素与向心性肥胖更相关。
Objective to explore the relationship between environment factors induced visceral obesity and insulin resistance.
目的探讨环境因素引起的内脏性肥胖与胰岛素抵抗的关系。
And it really is only visceral obesity - the amount of fat that you carrying around in the belly - and that really is the type of fat that is the bad fat.
而实际上只有内脏肥胖，即位于我们腹部的脂肪，实际上才是真正的坏脂肪。
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