Patients with this more advanced prostate cancer receive a therapy called androgen ablation, which inhibits production of testosterone - the culprit that allows a tumor to keep growing.
患有这类晚期前列腺癌的患者会接受一种叫做雄激素阻断的治疗,该治疗能够抑制睾酮的产生,睾酮被认为是肿瘤生长的帮凶。
As Thompson began to study the gene, he found that it was a target for a tumor suppressor gene called p53, which is a major controller of cell activity in prostate and other cancers.
当Thompson开始研究这个基因时,他发现RTVP - 1基因是肿瘤抑制基因p 53的靶基因,p 53是前列腺癌及其他癌症细胞活性的最主要控制者。
When they tested it on tumor cells in the lab, they found it fueled the growth of prostate cancer cells more potently than testosterone alone, they reported in the journal Clinical cancer Research.
当在实验室中用该产品在肿瘤细胞中进行试验时,他们发现与单独使用睾酮相比,该产品能更有力的促进前列腺癌细胞的生长。他们在《临床肿瘤研究》杂志上报道了这一研究。
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