Conclusion The rat model of depression has been established successfully. Chronic fluoxetine or tianeptine treatment significantly increases the neurogenesis in the rat hippocampus of depression model.
结论孤养与慢性不可预见性中等强度应激(CUMS)联合成功建立了抑郁模型;氟西汀和噻奈普汀的长期治疗都能促进抑郁模型大鼠海马神经发生,神经发生在抑郁症的治疗中可能具有重要作用。
参考来源 - 抗抑郁药物对抑郁模型大鼠海马神经发生的作用Material and Methods:Rats were divided into control group, stress group, stress-tianeptine group and 12 days after cessation stress group.
材料和方法:动物按体重随机分为对照组、应激组、应激+达体朗组、应激后12天组。
参考来源 - 慢性应激引起大鼠海马CA3区锥体细胞和血清皮质酮浓度的变化及相关研究·2,447,543篇论文数据，部分数据来源于NoteExpress
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