Objective: To research on doparainegic neuron synapse plasticity and its celluar molecular mechanism of substantia nigra in Parkinson’ s disease model mouse treated by electroacupuncture therapy.
目的：研究电针对帕金森小鼠黑质致密部多巴胺神经元突触可塑性的影响及细胞分子机制。
参考来源 - 电针促进帕金森小鼠多巴胺神经元突触可塑性的细胞分子机制·2,447,543篇论文数据，部分数据来源于NoteExpress
Objective To explore the mechanism, material fundamental of synapse plasticity and acupuncture's efficacy by watching the alteration of SYP after cerebral infarction and acupuncture treatment.
目的观察大鼠脑梗死及电针干预后突触素(syp)的动态变化，探讨突触可塑性的物质基础、机制及针刺的影响。
As a gas message molecule, nitric oxide takes part in the information transfer in nervous system, which affects plasticity of synapse in hippocampus, and cerebellum and construction of neural network.
一氧化氮作为一种气体信使分子，参与神经系统的信息传递，对海马、小脑等神经元突触的可塑性和神经网络的构建产生重要影响。
The damage of hippocampus neuron is the key part in plasticity regulation of synapse and play a critical role in the mechanism of depression.
海马神经元损伤是神经突触可塑性调节中重要的一个环节，在抑郁症发病机制的研究中占有重要地位。
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