Oncogenes are mutations that cause an excessive stimulation of the cell cycle, while mutations in tumor suppressor genes can result in a loss in function in controlling this excessive activity.

原癌基因的突变导致的过度刺激细胞周期，而突变的肿瘤抑制基因，会导致在这一功能的控制过度活动。

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As Thompson began to study the gene, he found that it was a target for a tumor suppressor gene called p53, which is a major controller of cell activity in prostate and other cancers.

当Thompson开始研究这个基因时，他发现RTVP - 1基因是肿瘤抑制基因p 53的靶基因，p 53是前列腺癌及其他癌症细胞活性的最主要控制者。

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