To understand further the molecular mechanism of its function, human neuroblastoma SK N SH cell line was chosen as a model to study its signal transduction pathway.

为了进一步阐明其作用的分子机制，以SK N SH成神经瘤细胞（SK细胞）为模型进行了研究。

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AIM to investigate the effect of mipafox that induced delayed neuropathy on neural differentiation in human neuroblastoma SK-N-SH cells and elucidate its mechanism.

目的研究诱发迟发性神经病的氟丙胺磷对人成神经细胞瘤sk - N - SH细胞分化的影响及其作用机理。

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Objective To explore the molecular mechanism of MMT-induced apoptosis in SK-N-SH cells.

目的研究甲基环戊二烯羰基锰（MMT）诱导人SK-N-SH细胞凋亡的分子机制。

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