Several studies have explored the role of S1P in the pathogenesis of RA.

已有几项研究阐述了1-磷酸鞘氨醇在类风湿关节炎发病中所起的作用。

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HGF stimulation could increase the activity of SPK and cellular S1P in ECV304 cells.

肝细胞生长因子(HGF)刺激能增强ECV304细胞SPK的活性和细胞内s1p水平。

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The combined tumorigenic and angiogenic effects of S1P make it an excellent target for anticancer therapy.

其致瘤和促血管生成的双重作用使得S1P成为一个作为抗癌治疗非常好的靶点。

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