ObjectiveTo establish a ideal experimental rat model of acute necrosis pancreatitis (ANP).

目的建立一种理想的急性坏死性胰腺炎（ANP）大鼠模型。

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Conclusion OXPAPC blocked LPS signal pathway, thus decreased the severity of experimental ANP in rat.

结论OXPAPC可阻断内毒素信号通路，从而显著降低实验性anp的严重程度。

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Methods The rat model of ANP was established by retrograde bile-pancreatic duct injection of 5% sodium taurocholate.

方法逆行胆胰管注射5 %牛磺胆酸钠溶液制成ANP模型。

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