CONCLUSION: the increase of neuron EGFR expression can be induced by focal cerebral ischemia injury, and may be related to the self protection of ischemia neurons.
结论：局灶性脑缺血损伤可诱导神经元egfr表达增加，EGFR表达增加可能与缺血神经元自身保护作用有关。
Objective to comparatively observe the changes of three-dimension topography in culture neuron membrane respectively by NMDA injury, MK-801 protection and normal cultivation.
目的对比观察正常培养、NMDA损伤及MK - 801保护皮层神经元膜表面三维构象形貌改变。
Conclusion: Cerebrolysin has the protection and treatment effect on HIE. The mechanism of the effect is correlated with blocking abnormal metabolism of no and reducing apoptosis of neuron.
结论：脑活素对HIE具有防治作用，其机理与阻断NO异常代谢、降低细胞凋亡有关。
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