CONCLUSION: High concentration of glucose inhibited endothelium-dependent vasorelaxation and this effect was unlikely mediated through activating oxygen-derived free radical production.
结论:高浓度葡萄糖抑制内皮依赖的血管舒张,这种作用不是通过增加氧自由基的产生介导的。
During the reperfusion period, endothelial dysfunction, activation of endogenous enzymes, leucocyte recruitment and activation all lead to the generation of oxygen -derived free radical.
再灌注期间,上皮细胞功能障碍,内源酶激活,白细胞聚集并活化,都会导致氧自由基的产生。
Conclusions the use of anisodamine before reperfusion could reduce the production of oxygen derived free radical, therefore alleviate the injury of grafted liver caused by ischemia reperfusion.
结论再灌流前应用山茛菪碱能减少再灌流后氧自由基的产生,可减轻缺血—再灌流对移植肝脏的损伤。
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