CONCLUSION: Captopril exerts electrophysiologic action due to decreasing delay outward rectification potassium current and prolonging action potential duration of APD30, APD50 and ERP.
结论：卡托普利具有的电生理作用是由于它降低外向延迟整流钾电流及延长APD30、APD50和ERP。
AIM: to evaluate the effect of captopril on action potential duration and outward delayed rectification potassium current (Ik).
目的：探讨卡托普利对豚鼠心肌细胞动作电位间期及延迟整流钾电流的作用。
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