Objective to observe the role of mitochondria dysfunction in neuron apoptosis during ischemia-reperfusion injury.
目的观察线粒体功能失调在缺血再灌后神经元凋亡中的作用。
One of the signs of ageing is weakening of skeletal muscles and increasing dysfunction of their mitochondria.
年老迹象之一是，骨骼肌的衰减及其线粒体的功能不良。
The dysfunction of mitochondria involves in the pathogenesis of shock and MODS.
线粒体功能紊乱参与了休克和多器官功能不全的发生。
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