The lithium-pilocarpine induced SE was used as the SE model.
建立氯化锂-匹罗卡品大鼠SE模型。
Conclusions PTZ -induced se can increase the dentate granule cell neurogenesis in the developing rat. NMDAR plays an important role in neurogenesis following seizures.
结论癫癎发作可增加幼鼠齿状回颗粒细胞的神经发生，而NMDAR在癫癎后的神经发生中起着促进作用。
RESULTS: Neuron necrosis, apoptosis, increase of apoptosis protein expression and proliferation of glial cells were the characteristics of pathological injury in the pilocarpine induced se models.
结果：毛果芸香碱惊厥持续状态模型鼠病理损伤特点为神经元坏死、细胞凋亡、凋亡蛋白表达增多，胶质细胞增生。
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