Previous study show that the infection viral and genetic sensitivity maybe the important initiating agent, PM is T cell mediated, DM is humoral immunity mediated autoimmunity disease. IBM is concerned with immunologic abnormality and myofibrosis.
既往研究已初步证实病毒感染及遗传易感性可能是IIM发病的重要始动因素,PM是T细胞介导的、DM是体液免疫介导的自身免疫性疾病,IBM与免疫异常及肌纤维变性有关。
参考来源 - 特发性炎性肌病的免疫病理机制研究进展·2,447,543篇论文数据,部分数据来源于NoteExpress
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