We investigated the dynamic changes of myocyteapoptosis and the expression of bcl-2 -. bax and Fas protein in ventricle andatrium of chronic pressure overload, using in situ terminal deoxynucleotidyltransferase mediated dUTP nick end labeling(TUNEL) and strept avidin-biotin-enzyme complex(SABC) immunohitochemistry.
采取末端脱氧核苷酸转换酶介导的dUTP-生物素平移末端标记(TUNEL)技术和链酶亲和素-生物素-过氧化物酶复合物(SABC)免疫组化方法,观察持续压力超负荷下兔各房室心肌细胞凋亡的动态变化及bcl-2、bax和Fas蛋白的表达。
参考来源 - 细胞调亡在心脏重塑中的作用及其调节机制·2,447,543篇论文数据,部分数据来源于NoteExpress
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