CONCLUSION: the up-regulation of the expression of hippocampal GAD67 protein in rats with temporal lobe epilepsy is one of the mechanisms of organic endogenous anti-epilepsy after seizures.
结论：颞叶癫痫大鼠海马gad67蛋白表达的增高是癫痫发生后机体的内源性抗痫机制之一。
Although prolonged febrile seizure is a risk factor of temporal lobe epilepsy, it is not clear whether febrile seizure provokes hippocampal abnormalities.
尽管长时间的热性惊厥是颞叶癫痫的危险因素，但是还是不清楚热性惊厥是否诱发了海马的异常改变。
4, Abnormal T2 signalwas present in the adjacent temporal lobe, amygdala, or insula in 27% of the 22children with abnormal hippocampal T2 signal compared with none of thecontrols.
在22名出现海马区异常信号的儿童中，有27%的儿童异常的T2信号出现在相邻的颞叶、杏仁核或岛叶。
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