粒细胞巨噬细胞集落刺激因子(granulocyte-macrophage colony-stimulating factor,GMCSF)是来源于创面修复细胞的4螺旋束原型结构细胞因子[3-5],与创面愈合密切相关。
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Half of the mice were injected with GMCSF, the other with a placebo saline solution.
一半的小白鼠注射GMCSF，而另一半则注射安慰剂生理盐水。
Conclusions GMCSF-absence inhibits neovascularization during wound repair and leads to the delay of wound healing.
结论GMCSF基因缺失影响创面的新生血管化能力，进而影响创面的愈合。
Conclusion GMCSF can up-regulate the expression of VEGF in human skin fibroblasts, and ERK pathway plays an important role in this process.
结论GMCSF可促进人皮肤成纤维细胞VEGF表达，ERK通路在此过程中发挥重要作用。
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