CONCLUSIONS: Coagulation cascade is activated and crosslinked fibrin forms in gallbladder bile before gallstone formation.
结论：胆石形成前即有凝血活化和交联纤维蛋白形成。
Conclusion: Plasma Fbg concentration and fibrin monomer polymerize function were decrease, showed that patients were low coagulation state and contributing to one cause of hemorrhage.
结论：失代偿性肝硬化患者因纤维蛋白原浓度下降致使纤维蛋白单体聚合功能降低，从而出现低凝状态，这可能是构成出血倾向的原因之一。
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