Fibroblast growth factor(FGF-1)lacks amino-terminal signal peptide, so it can't release through the classical endoplasmic reticulum(ER)-Golgi pathway.

酸性成纤维生长因子-1（FGF-1）由于氨基端缺乏信号肽序列，不能通过经典的内质网-高尔基体途径释放。

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ObjectiveTo evaluate the correlation between the promoter polymorphism of Fibroblast Growth Factor 1 (FGF-1) and late-onset Alzheimer's disease (LOAD).

目的探讨成纤维细胞生长因子1（FGF-1）基因启动子多态性是否与晚发型阿尔茨海默病（LOAD）相关。

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FGF-1 is involved in many pathological processes, so the measures to disturb or inhibit FGF-1 release pathway provide a new strategy for the diseases directed by FGF-1.

FGF - 1与许多病理过程有关，因而一些干预FGF - 1释放途径的措施为治疗FGF - 1介导的疾病提供了新的思路。

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Saturated fats too, the body triggers FGF-21 that blocks IGF-1 when the body burns fats to slow down the metabolism.

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