Objective To explore the mechanism of formaldehyde(FA)inhalation induced lung injury.

目的探讨甲醛中毒的肺脏损害机制。

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Recently, it was proposed that endogenous formaldehyde(FA) may be involved in endothelial damage, and may be a potential factor of vulnerability of atherosclerosis.

近年来，有研究提出内源性甲醛（FA）可能是启动内皮细胞损伤，导致动脉粥样硬化的一个潜在危险因素。

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