CONCLUSIONS: The content of NOS and NO induced by IL-1βincreased in the supernatant of pancreatic islet cells. Partial extracellular calcium ion influxed toβ-cell via voltage-gated calcium channel, inducing calcium overload in pancreatic β-cell.
结论：IL-1β可诱导胰岛β细胞内NOS活性增强，使NO合成增加，引起细胞外钙离子沿L型电压门控钙通道内流入细胞内，导致胰岛β细胞内钙超载，损伤胰岛β细胞正常功能。
参考来源 - IL·2,447,543篇论文数据，部分数据来源于NoteExpress
It is concluded that high extracellular calcium can injure renal tubular cells, but not by the way of destroying the plasma membrane integrity.
这些结果说明，细胞外高钙能够直接损害肾小管上皮细胞，但不是通过损伤细胞膜的方式来实现的。
Contracture occurs when the efflux of calcium is prevented by removing the extracellular sodium.
如果除去细胞外液的钠而阻止钙外流，就会出现肌肉挛缩了。
And 1% in parenchyma, extracellular fluid and blood, called the Immiscibility calcium pool.
其余1%的钙存在于软组织，细胞外液及血液中，称之为混溶钙池。
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