AIM To study on the alterations of t suppressor cell function in patients with ulcerative colitis (UC) and Crohn's disease (CD) and their clinical significance.

目的研究溃疡性结肠炎(UC)和克隆病(CD)患者T抑制细胞的活性及其临床意义。

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Oncogenes are mutations that cause an excessive stimulation of the cell cycle, while mutations in tumor suppressor genes can result in a loss in function in controlling this excessive activity.

原癌基因的突变导致的过度刺激细胞周期，而突变的肿瘤抑制基因，会导致在这一功能的控制过度活动。

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