Objective To investigate the changes of the expression of CPP32 and P53 protein following neuronal cell injury after focal cerebral ischemia in rats.
目的探讨大鼠脑缺血后神经元损害过程中CPP32和P5 3蛋白表达的变化。
Conclusion in the process of focal cerebral ischemia and reperfusion, regulating the quantity of NO in the propriety range may protect and avoid purpose of neuronal cell injury.
结论局灶性脑缺血后，调节NO生成量在适当范围，可以达到保护、防止神经细胞进一步损伤的目的。
Brain injury may lead to neuronal cell necrosis.
脑损伤可导致神经细胞坏死。
应用推荐