Conclusion The activity of the histaminergic system decreased and the decrease might be one of the compensatory mechanisms for maintenance of the normal sleep in the null mice lacking DPR.
结论DPR基因敲除型小鼠脑中组胺能神经活性显著降低，这一变化可能是该种小鼠尚能维持正常睡眠的代偿机制之一。
The proposed RMC solution is based on a layered architecture and a set of RMC mechanisms allowing for distributed development and for minimizing redundancy, inconsistency, and maintenance cost.
提议的RMC解决方案是基于分层的架构和一组虑及分布的开发和最小化冗余、不一致性，及维护成本的RMC机制的。
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