Conclusions Aging could accelerate fibrin deposition in pulmonary tissue of LPS-induced rat model of acute lung injury, and promote the injury of endothelial cell function caused by fibrin deposition.
结论增龄能够促进脂多糖诱导的急性肺损伤大鼠肺组织纤维蛋白沉积，增强纤维蛋白的内皮细胞损伤作用。
Her study was supported by the National Institute on Aging, National Heart Lung and Blood Institute and National Institute of Arthritis and Musculoskeletal and Skin Diseases.
她的研究得到国家老龄研究所，国家心肺和血液研究所和国立关节炎及肌骨及皮肤疾病研究所的支持。
Objective To research the relationship between dynamic lung function and aging in old people.
①目的探讨老年人动态肺功能与衰老的关系。
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