Objective To investigate the protective effect of nitric oxide (NO) induced by ischemic preconditioning (IP) on ischemic reperfusion injury of rat liver graft.
目的探讨一氧化氮（NO）在缺血预处理（IP）保护大鼠移植肝脏缺血再灌注损伤中的作用。
This article reviews the recent progress in er stress and UPR induced by cerebral ischemic-reperfusion.
文章对脑缺血再灌注诱导内质网应激和upr的研究进展做了综述。
Lidocaine attenuates cell injury induced by ischemic-reperfusion and inflammation, although the protective mechanisms are not understood.
利多卡因可减轻缺血再灌注和炎症反应所致的细胞损伤，其机制至今不明。
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