The bottom of the gut origin infection s pathophysiology is intestinal mucous membrane barrier dysfunction.
肠源性细菌感染的病理生理学基础,是肠黏膜屏障功能障碍。
参考来源 - 尿激酶对大鼠急性重症胰腺炎肠道微循环障碍的作用·2,447,543篇论文数据，部分数据来源于NoteExpress
Mitochondria dissolved into vacuole in intestinal mucous membrane epithelial cells.
肠上皮细胞的线粒体空泡化。
Subsequently, Intestinal Mucous Membrane Microvascular Cells (IMECs) were cultivated.
随后进行大鼠肠黏膜微血管内皮细胞的培养。
Whether nitrogen monoxide affect on iron metabolism in intestinal mucous membrane or not, the first pace of iron transport, should be researched deeply.
小肠黏膜上皮是机体铁转运的第一步，一氧化氮是否影响此处铁的转运有待于进一步分析。
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