The inhibition of product exists during ethanol production by fermentation when the concentration of bio-ethanol product reaches to a certain level.
在乙醇发酵生产过程中，当产物积累到一定程度时，存在产物抑制现象。
Conclusion: Our results suggest that long-term stimulus with the low-concentration ethanol-induced gastric mucosal cell proliferation is related with BMK1 signaling pathway via ROS production.
结论：慢性饮酒刺激胃黏膜底腺干细胞的增殖可能与酒精引起活性氧（ROS）增多有关，同时本研究还第1次发现慢性饮酒诱导胃底腺干细胞增殖是由BMK1所介导。
In YPD media with 0.1% levoglucosan, the enhancement of cell concentration and ethanol production was approximately 14.3% and 25.4%, respectively.
在YPD培养基中添加0.1%内醚糖，细胞浓度和乙醇产率最高，其中细胞浓度提高14.3%，乙醇产率提高25.4%。
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