Conclusion Propofol may inhibit hypoxia in the brain and the apoptosis of nerve cells in result of protecting the cerebral ischemia and reperfusion damage in rats.
结论异丙酚可能通过抑制大脑缺氧，抑制神经元的凋亡，从而对大鼠局灶性脑缺血-再灌注损伤发挥保护作用。
Objective To investigate the transcription-coupled repair factor CSB/ERCC6 expression and neuronal DNA damage in ischemic area after cerebral ischemia-reperfusion in rats.
目的探讨大鼠脑缺血再灌注后缺血区不同时相转录修复偶联因子（CSB/ERCC6 ）表达对神经元DNA损伤的作用。
Hyperglycemia significantly increases brain damage, mortality rate, and long-term disability after cerebral ischemia and reperfusion injury.
高血糖症会显著增加脑损伤，导致病死率升高，并由脑缺血再灌注损伤后导致患者长期残疾。
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