Such watershed infarctions can occur with relative or absolute hypoperfusion of the brain.

相对或绝对的脑低灌注都可能造成这种梗死。

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There were obvious pathological change in the brain after the chronic cerebral hypoperfusion, such as myelinic degeneration and formation of glial nodule in white matter.

缺血后脑的病理改变以神经细胞变性、固缩和退变，皮层下白质的神经纤维和髓鞘溃变为主，普遍存在胶质细胞增生，形成胶质小结。

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Objective To investigate the effects of activation of leukocytes on brain damages after chronic cerebral hypoperfusion.

目的探讨白细胞的活动在慢性脑灌注不足脑损害中的作用。

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