首先，他们用基因手段禁止了细胞受体的活动——细胞受体即那些向其他细胞发送和从其他细胞接受信号的分子们——因为雌激素和雄激素存在于正在发育的老鼠胚胎的肢芽中。

First, they used genetic tools to deactivate cell receptors-molecules that send and receive signals to and from other cells-for testosterone and estrogen in the developing limb buds of mouse embryos.

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此外，在ER+/PR+（雌激素受体阳性/孕激素受体阳性）恶性肿瘤和接受阿那曲唑作为内分泌治疗的患者中，发现了（大豆异黄酮和绝经妇女乳腺癌复发率之间）相反的关联。

Additionally, inverse associations were found in those who wee diagnosed with ER+/PR+ malignancy and those who receiving anastrozole as endocrine therapy.

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