许多基因有助于防止肿瘤的生长，但表观基因组遭到破坏时，可能会碰巧把这些具有保护作用的基因切换到关闭状态。
Many genes help to prevent tumours from growing, but disruption of the epigenome can switch these protective genes off by accident.
然而，仅靶向作用于VEGF蛋白在黑色素瘤病人中并不能防止肿瘤的生长。
However, targeting VEGF alone does not prevent tumor development in melanoma patients.
同样，在卵巢癌小鼠模型中，抑制应激和相关的去甲肾上腺素和肾上腺素增加可防止肿瘤细胞发生失巢凋亡，从而促进肿瘤生长。
Similarly, in a mouse model of ovarian cancer, restraint stress and the associated increases in norepinephrine and epinephrine protected the tumor cells from anoikis and promoted their growth.
应用推荐