方法：腹腔注射(ip)D-gal150mg·kg^-1，每天1次．连续56天．诱导胰岛素抵抗(insdin resistance，IR)的IGT大鼠模型；观察D-gal诱导大鼠空腹血糖(fasting blood glucose。
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结论：高脂饲料和高果糖餐喂饲均可以诱导胰岛素抵抗大鼠模型。
CONCLUSION: high fat feed and high laevulose forage can both induce the insulin resistance in rats.
目的探讨高脂饲料诱导胰岛素抵抗(IR)动物模型的内皮功能变化及其机制。
Objective to observe the change of endothelium-dependent vasodilatation function and to explore its mechanism in insulin-resistant (IR) rats induced by high fat feed.
本文主要针对活性氧的产生、氧化应激诱导胰岛素抵抗和胰岛细胞功能受损等机制进行了综述。
In this paper, we summary the mechanism of the ROS generation, insulin resistance and cell dysfunction induced by oxidative stress.
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