诱导的大鼠急性肺损伤 acute lung iniury ; ALI
HMGB1在脂多糖诱导的急性肺损伤(ALI) P MN凋亡改变中有重要的调节作用。
HMGB1 may play an important role as a modulator in apoptotic changes in PMN during LPS-induced ALI.
目的：探讨己酮可可碱（PTX）对内毒素（L PS）诱导的急性肺损伤肺微血管通透性的影响。
Objective:To study the effects of pentoxifylline (PTX) on pulmonary microvascular permeability of LPS induced acute lung injury.
结论增龄能够促进脂多糖诱导的急性肺损伤大鼠肺组织纤维蛋白沉积，增强纤维蛋白的内皮细胞损伤作用。
Conclusions Aging could accelerate fibrin deposition in pulmonary tissue of LPS-induced rat model of acute lung injury, and promote the injury of endothelial cell function caused by fibrin deposition.
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