血浆肾素活性,饮食同醛固酮要求,RIA法:0.82~2.0nmol/(L?h)。
事实上,基础ANP及基础血浆肾素活性(PRA)是正常的,并与疾病的活性及高血压无关。心肌肥厚将导致心脏的功能改变,心脏的收缩功能在静止的状态下是正常的,但在劳累时出现...
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Plasma renin activity (PRA) and Ang Ⅱin 2 groups statistically reduced (P<0.05) and LVEF in 2 groups statistically significantly increased (P<0.01) after treatment. LVESD and LVESV in carvedilol group statistically decreased (P<0.05) after treatment.
2组于治疗后血浆肾素活性(PRA)、血管紧张素Ⅱ(AngⅡ)浓度较治疗前降低(P<0.05),左室射血分数增加(P<0.01)。
参考来源 - 长期应用美托洛尔和卡维地洛联合血管紧张素转换酶抑制剂对心肌梗死患者左室重构及心功能的影响·2,447,543篇论文数据,部分数据来源于NoteExpress
当联合使用一种ACE抑制剂时,血浆肾素活性增强是如此之大。
When you combine an ACE inhibitor b, the plasma renin activity increases so much.
目的:分析血浆肾素活性(PRA)、血浆血管紧张素原(AO)浓度与原发性高血压的关系。
Objective:To investigate the relationship among essential hypertension, plasma renin activity (PRA) and angiotensinogen(AO).
与肾实质疾病患者相比,肾动脉狭窄患者在开搏通试验后血浆肾素活性明显升高,而血清醛固酮显著下降。
Comparing with renal parenchyma hypertension, plasma renin activity increased significantly after Capoten test in renal artery stenosis and serum aldosterone level decreased significantly.
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