结果提示：（1）在病毒感染中肝血管收缩反应性上升及血小板粘附功能增强是肝微循环障碍发生的基础。
The results show; (1) The increased hepatic vasoconstrictor reactivity and the adhesive function of platelets in viral infection are the basic mechanism of the hepatic microcirculatory disturbance.
结果：二甲基亚砜组血小板粘附率、聚集功能和低渗反应回复率均较对照组和洗涤血小板组降低。
RESULTS: in the DMSO group, platelet adherence rate, aggregating function, and recovery rate from hypotonic shack decreased during storage comparing with control and washed platelet groups.
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