应用扫描镜超微病理技术,分析对比了肺癌患者胸膜间皮细胞与正常人胸膜间皮细胞间超微形态的异同。
The ultrastructures of pleural mesothelioma were compared between patients patients with lung cancer and normal subjects by pathological technique under scanning electron microscope.
目的研究前炎症细胞因子对人胸膜间皮细胞(HPMC)释放纤溶酶原激活物抑制剂(PAI 1)的诱导效应。
Objective To investigate effects of two proinflammatory cytokines on the production of plasminogen activator inhibitor 1(PAI 1) by human pleural mesothelial cells(HPMC).
结论:胸膜间皮细胞存在AQP - 1表达,地塞米松对胸膜间皮细胞AQP - 1表达有明显的增强性调节作用,具有时间依赖性。
CONCLUSION: the expression of AQP-1 in cultured rat pleural mesothelial cells is confirmed at the protein and mRNA level. Dex increases the expression of AQP-1 in a time-dependent manner.
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