背景提高有缺陷的囊性纤维化跨膜电导调节(CFTR)蛋白的活性是囊性纤维化的一种潜在治疗。
BACKGROUND Increasing the activity of defective cystic fibrosis transmembrane conductance regulator (CFTR) protein is a potential treatment for cystic fibrosis.
首先，基于紧束缚模型研究了非磁性点缺陷对金属型单壁碳纳米管态密度和电导的影响。
Firstly, the influence of point defects on the density of states and conductance for metallic carbon nanotubes is studied based on the tight-binding model.
光照时电导率增加，光注入后因光生载流子对缺陷态的填充使费米能级上移，从而使激活能减小；
Their Fermi levels moved upward as the defect states were filled by light generated carriers. As a result, their activation energies decreased.
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